Rosiglitazone treatment prevents mitochondrial dysfunction in mutant huntingtin-expressing cells - Possible role of peroxisome proliferator-activated receptor-gamma (PPAR gamma) in the pathogenesis of Huntington disease

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dc.contributor.authorQuintanilla Gómez, Rodrigo Arthur
dc.contributor.authorFuenzalida Morales, Karen Andrea
dc.contributor.authorBronfman A., Miguel L.
dc.date.accessioned2017-04-25T19:28:15Z
dc.date.available2017-04-25T19:28:15Z
dc.date.issued2008
dc.identifier.issn0021-9258
dc.identifier.urihttps://repositorio.uc.cl/handle/11534/20868
dc.language.isoen
dc.nota.accesoContenido completo
dc.relation.isformatofJournal of Biological Chemistry Vol. 283, no. 37 (2008), p. [25628]-25637
dc.revistaJournal of Biological Chemistryes_ES
dc.rightsacceso abierto
dc.subject.ddc610
dc.subject.deweyMedicina y saludes_ES
dc.subject.otherEnfermedad de Huntington - Patologíaes_ES
dc.subject.otherMitocondrias - Metabolismoes_ES
dc.subject.otherPPAR gammaes_ES
dc.titleRosiglitazone treatment prevents mitochondrial dysfunction in mutant huntingtin-expressing cells - Possible role of peroxisome proliferator-activated receptor-gamma (PPAR gamma) in the pathogenesis of Huntington diseasees_ES
dc.typeartículo
sipa.codpersvinculados3175
sipa.codpersvinculados98819
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