Increased diagnosis of primary aldosteronism, including surgically correctable forms, in centers from five continents

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dc.article.number106217
dc.contributor.authorMulatero, P
dc.contributor.authorStowasser, M
dc.contributor.authorLoh, KC
dc.contributor.authorFardella, CE
dc.contributor.authorGordon, RD
dc.contributor.authorMosso, L
dc.contributor.authorGomez Sanchez, CE
dc.contributor.authorVeglio, F
dc.contributor.authorYoung, WF
dc.contributor.otherNCD Risk Factor Collaboration (NCD-RisC)
dc.date.accessioned2024-08-15T08:00:25Z
dc.date.available2024-08-15T08:00:25Z
dc.date.issued2004
dc.description.abstractRecently, some genetic forms of hypertension have been well characterized. These forms can be globally called mineralocorticoid hypertension and are due to different alterations of the renin-angiotensin-aldosterone system (SRAA). Among these, classic primary hyperaldosteronism and its glucocorticoid remediable variety, in which hypertension is secondary to aldosterone production, must be considered. There are also conditions in which mineralocorticoid activity does not depend on aldosterone production. These conditions generate a hyporeninemic hyperaldosteronism, observed in Liddle syndrome, apparent mineralocorticoid hypertension, 11- and 17-hydroxilase deficiency, among others. The detection of these forms of hypertension is only feasible if the renin-angiotensin-aldosterone system is assessed, measuring renin and aldosterone levels. This article reviews these forms of hypertension, their clinical workup and their relevance in the usual hypertensive patients.
dc.description.funderANID through FONDECYT
dc.format.extent1 página
dc.fuente.origenWOS
dc.identifier.doi10.1097/00004872-200406002-00629
dc.identifier.eissn1524-4563
dc.identifier.issn0263-6352
dc.identifier.pubmedidMEDLINE:10451632
dc.identifier.scopusidSCOPUS_ID:0346985951
dc.identifier.urihttps://doi.org/10.1097/00004872-200406002-00629
dc.identifier.urihttps://repositorio.uc.cl/handle/11534/87472
dc.identifier.wosidWOS:000223099400627
dc.information.autorucFacultad de Medicina; Mosso Gomez, Lorena Montserrat; S/I; 88201
dc.issue.numero5
dc.language.isoen
dc.nota.accesoSin adjunto
dc.pagina.final610
dc.pagina.inicio604
dc.relation.ispartof14th European Meeting on Hypertension, JUN 13-17, 2004, Paris, FRANCE
dc.revistaREVISTA MEDICA DE CHILE
dc.rightsregistro bibliográfico
dc.subjecthypertension
dc.subjecthyperaldosteronism
dc.subjectrenin-angiotensin system
dc.subjectLiddle syndrome
dc.subjectGLUCOCORTICOID-REMEDIABLE ALDOSTERONISM
dc.subjectSEVERE 17-ALPHA-HYDROXYLASE DEFICIENCY
dc.subjectPRIMARY HYPERALDOSTERONISM
dc.subjectLIDDLES SYNDROME
dc.subjectSALINE INFUSION
dc.subjectACTIVITY RATIO
dc.subjectDIAGNOSIS
dc.subjectREABSORPTION
dc.subjectVOLUNTEERS
dc.subjectP450C11AS
dc.subject.ddc550
dc.subject.deweyCiencias de la tierraes_ES
dc.titleIncreased diagnosis of primary aldosteronism, including surgically correctable forms, in centers from five continents
dc.typecomunicación de congreso
dc.volumen22
sipa.codpersvinculados88201
sipa.indexWOS
sipa.trazabilidadCarga WOS-SCOPUS;15-08-2024
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