Síndrome de respuesta inflamatoria sistémica severa: ¿Es comparable a la sepsis severa?

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dc.contributor.authorHernández P., Glenn
dc.contributor.authorDougnac Labatut, Alberto
dc.contributor.authorCastro O, J.
dc.contributor.authorLabarca M, E.
dc.contributor.authorOjeda M, M.
dc.contributor.authorBugedo Tarraza, Guillermo
dc.contributor.authorCastillo Fuenzalida, Luis Benito
dc.contributor.authorAndresen Hernández, Max
dc.contributor.authorBruhn, Alejandro
dc.contributor.authorHuidobro M, L.F.
dc.contributor.authorHuidobro M, R.
dc.contributor.authorCaballero G, M.T.
dc.contributor.authorHernández M, A.
dc.date.accessioned2023-06-30T20:48:42Z
dc.date.available2023-06-30T20:48:42Z
dc.date.issued1999
dc.description.abstractBackground: In 1992, a consensus conference defined the terms systemic inflammatory response syndrome (SIRS), sepsis, severe sepsis and septic shock. Since then, numerous reports have validated the prognostic usefulness of these operative definitions. Aim: To evaluate if sepsis severity criteria, as defined by the Consensus Conference, can be applied to noninfectious SIRS. Patients and methods: Five hundred eighteen patients admitted to 5 intensive care units (ICU) from 4 hospitals were prospectively evaluated during a 3 months period. Patients that met at least one severity criteria were included. SIRS etiology, organ dysfunction and evolution were recorded in each patient. Results: One hundred two patients were included: 79 with sepsis (group I) and 23 with noninfectious SIRS (group II). ICU and hospital mortality were comparable (43 and 48% in sepsis compared to 43 and 51% in non infectious SIRS). The most common sources of sepsis were pneumonia and peritonitis. Group II patients had a wide variety of diseases. ICU stay, APACHE score and number of organs with dysfunction were not different among groups. Only the incidence of renal dysfunction was higher in the septic group. Conclusions: The Consensus sepsis severity criteria can be applied to noninfectious SIRS, defining a population subset with similar high mortality and organ dysfunction incidence, although with greatly heterogeneous etiologies.
dc.fechaingreso.objetodigital2023-06-30
dc.format.extent6 páginas
dc.fuente.origenORCID
dc.identifier.doi10.4067/S0034-98871999001100007
dc.identifier.issn0034-9887
dc.identifier.urihttp://dx.doi.org/10.4067/S0034-98871999001100007
dc.identifier.urihttp://www.scopus.com/inward/record.url?eid=2-s2.0-0042539793&partnerID=MN8TOARS
dc.identifier.urihttps://repositorio.uc.cl/handle/11534/74044
dc.information.autorucEscuela de Medicina ; Hernández P., Glenn ; S/I ; 98874
dc.information.autorucEscuela de Medicina ; Dougnac Labatut, Alberto ; S/I ; 54554
dc.information.autorucEscuela de Medicina ; Bugedo Tarraza, Guillermo ; 0000-0001-7527-6202 ; 60490
dc.information.autorucEscuela de Medicina ; Castillo Fuenzalida, Luis Benito ; S/I ; 54397
dc.information.autorucEscuela de Medicina ; Andresen Hernández, Max ; 0000-0003-1874-1416 ; 54274
dc.information.autorucEscuela de Medicina ; Bruhn, Alejandro ; 0000-0001-8034-1937 ; 741
dc.issue.numero11
dc.language.isoes
dc.nota.accesoContenido parcial
dc.pagina.final1344
dc.pagina.inicio1339
dc.revistaRevista médica de Chile
dc.rightsacceso abierto
dc.subjectInflammatory response
dc.subjectSepsis syndrome
dc.subjectShock, septic
dc.subjectSyndrome, systemic
dc.titleSíndrome de respuesta inflamatoria sistémica severa: ¿Es comparable a la sepsis severa?
dc.title.alternativeSystemic inflammatory response syndrome. Is it similar to severe sepsis?
dc.typeartículo
dc.volumen127
sipa.codpersvinculados98874
sipa.codpersvinculados54554
sipa.codpersvinculados60490
sipa.codpersvinculados54397
sipa.codpersvinculados54274
sipa.codpersvinculados741
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